Anatomy of ovarian disease

2021-05-12 05:59 PM

Inflammatory pathology is very rare. Tumours can arise from the complex tissue of an adult ovary or from a vestige of the embryonic tissue of the ovary.

The gonads appear in the first month of the fetus as a localized ridge on the front-inner side of the kidneys called the genital crest. The genital cyst is covered by the epithelium of the cavity and has a central region that cushions the fibres below the epithelium. By the sixth week, the primitive germs migrate from the embryo sac into the genital crest. At this time, the primitive cavity hyperplasia actively penetrates the stroma, forms primitive cell lines that slowly surround the germs. By the tenth week, the undifferentiated gonads gradually turn into ovaries. The medullary region atrophy, forming primitive follicles in the shell with the development of the secondary cord in the shell region. These lines break to form follicles around the germ cell,

In newborn babies, the ovaries are long and narrow, located in the real pelvis, size 1.3x0.5x0.3cm. At puberty, the ovaries are oval in shape, measuring 3x1.8x1.2cm. In adult women, the ovaries are larger: 3-5cmx1.5-3cmx 0.6-1.5cm, weighing 5-8 grams. After menopause, ovaries shrink. The shell is white, pink, smooth, yellow, white, scattered in the shell and marrow area of ​​the ovary.

The most common damage to the ovaries is an ovarian tumour. Inflammatory pathology is very rare. Tumours can arise from the complex tissue of an adult ovary or from a vestige of the embryonic tissue of the ovary. Cystic tumours are more common than solid tumours.

In terms of incidence, ovarian cancer ranks 6th, after cancer of the breast, colon, cervix, body of the uterus, and stomach, but until now, ovarian cancer is still the cause of gynaecological death. top in Europe. In the US, the number of new cases per year is 14.2 cases per 100,000 white women and 9.3 per 100,000 black women (Greene, 1984). This cancer has a high prevalence in Northern Europe and North America, and low in Japan (Gorins, 1985). In France, in 1982, the number of new cases was 10.6 per 100,000 women aged 35-64 years.

Among risk factors, there is family. Piver (1984) and Heintz (1985) found more than two families with ovarian cancer. Familial cancers usually occur earlier, and 90-97% of tumours are clear fluid and have many different types of poorly differentiated adenocarcinomas. Ovarian cancer is more likely to occur in women who have no birth or have few children. In contrast, many authors such as Cramer and Weiss have found that the incidence of ovarian cancer is low in women taking oral contraceptives long-term. Other risk factors such as dietary factors, high-fat content and low vitamin A or virus play a negligible role.