Pathophysiology prevention and treatment

2021-04-26 03:13 PM

An understanding of the role of causes and conditions will help in formulating the correct prevention plan.

One can cause an experimental disease and an experimental treatment before its clinical application, thus understanding the relationship between aetiology and pathogenesis, understanding the mechanism of disease arising, development and end. The physician knows when to be treated. There are many treatments such as symptomatic treatment, cause treatment, etc.

Symptomatic therapy is only used when diagnosing the cause of an unknown cause and when the manifestation of the disease is too strong that can affect the patient's life, such as excessive pain that can cause shock, high fever causing convulsions in children. Etc. However, should not be too abusive because sometimes more harm than good and just a countermeasure.

Treating the cause is best because it hits the causative factor. However, there are many diseases with unknown cause or as in many diseases with known causes, but when the disease develops, it may develop stronger, dangerous for the patient, it is necessary to use the concept of prevention in Treatment means adopting a certain course of pathology (pathophysiology of the disease) that sets out appropriate treatment measures to prevent or limit adverse events.

An understanding of the role of causes and conditions will help in formulating the correct prevention plan. The disease can be prevented by destroying and preventing the cause from developing, preventing conditions conducive to the cause (habitat, vectors of disease transmission, nutrition, etc.), enhancing the resistance of the disease. body.



Pathophysiology of cardiogenic shock

Urine formation: Reabsorbed glomerular filtration

Air in and out of the lungs: pressure causes the movement of air

Mechanism of urine concentration: osmotic pressure changes in different segments of the renal tubule

Absorption and excretion of potassium through the kidneys

Nephron: The functional unit of the kidney

Estimated renal plasma flow: PAH clearance

Prothrombin activation: initiates blood clotting

Pulmonary capillary dynamics: capillary fluid exchange and pulmonary interstitial fluid dynamics

Graphical analysis of high-volume heart failure

Red blood cells: differentiation and synthesis

Calculate the glomerular filtration rate (GFR): the forces that cause the filtration process

Ammonia buffering system: excretes excess H + and creates new HCO3

Concentrated urine formation: urea contributes to increased osmotic pressure in the renal medullary

Reduced sodium chloride, dilates arterioles, increases Renin release.

Extracellular fluid distribution between interstitial space and blood vessels

The proximal tubule reabsorption: active and passive reabsorption

Pathophysiology of fever

Origin of lymphocytes: the body's resistance to infection

Acidosis causes a decrease in HCO3- / H + in renal tubular fluid: compensation mechanism of the kidney

The endocrine regulates tubular reabsorption

Sodium channel blockers: decrease the reabsorption of sodium in the manifold

Self-regulation of glomerular filtration rate and renal blood flow

Physiological anatomy of the kidneys and urinary system

The myogenic mechanism itself regulates renal blood flow and glomerular filtration rate