Quadriplegic Fallot: congenital heart disease with left right shunt

2021-04-29 04:42 PM

Most of the reduced blood passes through the lungs, so the arterial blood is mostly undoxified venous blood. In this condition, four cardiac abnormalities occur simultaneously.

The Fallot quadriplegic is shown in the figure; it is the most common cause of "cyanosis. Most of the decreased blood passes through the lungs, so the arterial blood is mostly undoxified venous blood. In this condition, four cardiac abnormalities occur." out simultaneously:

1. The aorta originates in the right ventricle rather than the left, or it presses a hole in the septum, receiving blood from both ventricles.

2. Because the pulmonary artery is blocked, the amount of blood flowing from the right ventricle to the lungs is much lower than normal; instead, most of the blood goes directly to the aorta.

3. Blood from the left ventricle flows through a ventricular opening into the right ventricle and then into the aorta or directly into the aorta to press on this hole.

4. Since the right side of the heart has to pump large amounts of blood to counteract the high pressure in the aorta, its muscular system is highly developed, causing right ventricular hypertrophy.


Pathophysiology of cardiogenic shock

Urine formation: Reabsorbed glomerular filtration

Air in and out of the lungs: pressure causes the movement of air

Mechanism of urine concentration: osmotic pressure changes in different segments of the renal tubule

Absorption and excretion of potassium through the kidneys

Prothrombin activation: initiates blood clotting

Pulmonary capillary dynamics: capillary fluid exchange and pulmonary interstitial fluid dynamics

Graphical analysis of high-volume heart failure

Calculate the glomerular filtration rate (GFR): the forces that cause the filtration process

Estimated renal plasma flow: PAH clearance

Nephron: The functional unit of the kidney

Reduced sodium chloride, dilates arterioles, increases Renin release.

Ammonia buffering system: excretes excess H + and creates new HCO3

Red blood cells: differentiation and synthesis

Concentrated urine formation: urea contributes to increased osmotic pressure in the renal medullary

Extracellular fluid distribution between interstitial space and blood vessels

The proximal tubule reabsorption: active and passive reabsorption

Origin of lymphocytes: the body's resistance to infection

The endocrine regulates tubular reabsorption

Acidosis causes a decrease in HCO3- / H + in renal tubular fluid: compensation mechanism of the kidney

Sodium channel blockers: decrease the reabsorption of sodium in the manifold

Physiological anatomy of the kidneys and urinary system

Self-regulation of glomerular filtration rate and renal blood flow

Pathophysiology of fever

The kidneys excrete sodium and fluid: feedback regulates body fluids and arterial pressure